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PTSD Dx

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This page contains information and summaries of published research that are intended for people with professional training in mental health.  We urge caution since it may be difficult to know how to interpret the information without advanced training in psychology or psychiatry.

We recommend that readers interested in information intended for the general public check other areas of this web site (such as Info for Adults or Info for Children).  They contain similar information, but in a more readable form.  Also, elsewhere we have provided links to other websites that we feel may be helpful.

 

POSTTRAUMATIC STRESS DISORDER (PTSD)

AS A DIAGNOTIC ENTITY

   

The diagnostic criteria, as listed in DSM-IV, are:

POSTTRAUMATIC STRESS DISORDER (from DSM-IV, American Psychiatric Association)

Inclusion: A1 and A2

(A1) The person has been exposed to a traumatic event in which he/she experienced, witnessed, or was confronted with an event or events that involved actual or threatened death or serious injury, or a threat to the physical integrity of oneself or others.

(A2) The person's response to the traumatic event involved intense fear, helplessness, or horror.

 

Required: at least 1 from B

(B) The traumatic event is persistently re-experienced in at least 1 of the following ways:

(B1) Recurrent and intrusive distressing recollections of the event, including images, thoughts, or perceptions.

(B2) Recurrent distressing dreams of the event.

(B3) Acting or feeling as if the traumatic event were recurring (includes a sense of reliving the experience, illusions, hallucinations, and dissociative flashback episodes, including those that occur on awakening or when intoxicated.

(B4) Intense psychological distress at exposure to internal or external cues that symbolize or resemble an aspect of the traumatic event.

(B5) Physiological reactivity upon exposure to internal or external cues that symbolize or resemble an aspect of the traumatic event.

 

Required: 3 from C

(C) Persistent avoidance of stimuli associated with the trauma and numbing of general responsiveness (not present before the trauma), as indicated by at least 3 of the following:

(C1) Efforts to avoid thoughts, feelings, or conversations associated with the trauma.

(C2) Efforts to avoid activities, places, or people that arouse recollections of the trauma.

(C3) Inability to recall an important aspect of the trauma.

(C4) Markedly diminished interest or participation in significant activities.

(C5) Feeling of detachment or estrangement from others.

(C6) Restricted range of affect (e.g., unable to have loving feelings).

(C7) Sense of a foreshortened future (e.g., does not expect to have a career, marriage, children, or a normal life span).

 

Required: 2 from D

(D) Persistent symptoms of increased arousal (not present before the trauma), as indicated by at least 2 of the following:

(D1) Difficulty falling or staying asleep.

(D2) Irritability or outbursts of anger.

(D3) Difficulty concentrating.

(D4) Hypervigilance.

(D5) Exaggerated startle response.

 

Required: E and F

(E) Duration of the disturbance (symptoms in Criteria B, C, and D) is more than 1 month.

(F) The disturbance causes clinically significant distress or impairment in social, occupational, or other important areas of functioning.

 

 The three clusters of Sx (re-experiencing, numbing/avoidance, and arousal) that are currently used were derived by clinical observation rather than research (Foa & Rothbaum, 1998).

 

 

 

RESEARCH SUGGESTING MODIFICATIONS OF

THE PTSD DIAGNOSIS IN DSM-IV

 Since the publication of the DSM-IV criteria, empirical research has suggested that the diagnosis may need revisions for the next DSM, and that some of these revisions could have a substantive effect on treatment.

   

Subsequent factor analytic research has found that numbing and avoidance actually belong to different clusters (Foa et al., 1995). 

bulletFoa & Rothbaum (1998) indicate that this distinction is not just a matter of statistical niceties.  They suggest that effortful avoidance and numbing are two separate mechanisms (strategic versus more biological)

 

bulletFactors of increased irritability and anger belong with numbing (Foa & Rothbaum, 1998)
Prominent anger correlates with poorer treatment outcome in the available research, and theorists have suggested that anger may be used to inhibit or avoid symptoms of anxiety and thus prevent habituation upon exposure to a feared stimulus (Riggs et al., 1995)

   

Some researchers have also have noted that there are data that suggest that the 1 month time post-trauma criterion for PTSD should be reconsidered: 

bulletSome have suggested that the duration criterion should be eliminated, and PTSD should be used as a diagnosis soon after a trauma instead of Acute Stress Disorder since they have similar predictive power for later PTSD
bulletSpontaneous remission rates begin to flatten between 3 and 6 months. 
bulletDuring the first three months following a trauma people who develop PTSD show a progressive worsening of Sx over the first three months whereas people whose Sx remit show a gradual amelioration of Sx (Koren et al., 1999).

 

 

Sleep disturbances, aside from nightmares, may not be more common in people with PTSD than in others who have experienced traumatic incidents but who do not meet criteria for PTSD (Krakow et al, 20002; Klein et al., 2002).

 

 

Other Notes on Diagnosis:

Some of the DSM symptoms of PTSD appear to be particularly useful in distinguishing it from other disorders:

bulletflashbacks and nightmares are unique to PTSD (McFarlane, 1998b)
bulletvery few people without PTSD endorse numbing symptoms (Foa & Rothbaum, 1998)

 

 

Although the DSM indicates that “delayed onset” should be noted, research so far has found that there is not much empirical support for “delayed onset” categorization of PTSD. 

bulletMost reports of delayed onset PTSD in published studies have been anecdotal and retrospective (Foa & Rothbaum, 1998). 
bulletIn a prospective study of delayed onset PTSD, Bryant & Harvey (2002) found that people who had delayed onset of symptoms were characterized by elevated resting heart rates and psychopathology within a month of trauma, and elevated psychopathology at six months post trauma. 

Ø       These findings suggest that “delayed onset” may not occur without prior, significant symptoms

 

 

 

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The information posted on this site reflects our understanding of peer-reviewed research and generally accepted principles in psychology.  It is not intended to be used for self-treatment or as a substitute for individualized assessment and treatment by a licensed professional, and should not be construed as professional advice.

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